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Volume 122, Issue 8 , Pages 747-753 , August 2009

Diagnostic Criteria for Atrophic Rhinosinusitis

Tran H. Ly, MD
- Division of Clinical Immunology and Allergy, Department of Medicine, University of Mississippi Medical Center, Jackson
Richard D. deShazo, MD
- Division of Clinical Immunology and Allergy, Department of Medicine, University of Mississippi Medical Center, Jackson
- Requests for reprints should be addressed to Richard D. deShazo, MD, Department of Medicine, University of Mississippi Medical Center, 2500 North State Street, Jackson, MS 39216-4505
Jake Olivier, PhD
- NSW Injury Risk Management Research Centre, University of New South Wales, Sydney, Australia
- School of Mathematics and Statistics, University of New South Wales, Sydney, Australia
Scott P. Stringer, MD
- Department of Otolaryngology, University of Mississippi Medical Center, Jackson
William Daley, MD
- Department of Pathology, University of Mississippi Medical Center, Jackson
Christopher M. Stodard, MSc
- School of Medicine, University of Mississippi Medical Center, Jackson

Receiver operating characteristic curve demonstrating the relationship between the presence of the number of characteristics with sensitivity and specificity. Chronic nasal obstruction is excluded.

Receiver operating characteristic curve demonstrating the relationship between the presence of the number of characteristics with sensitivity and specificity. Chronic nasal obstruction is excluded.
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Rhinoscopic views in atrophic rhinosinusitis (A) through a nasal septum perforation shows surrounding crusting and bleeding. (B) The right nasal passage reveals a partial middle turbinectomy with dry

Rhinoscopic views in atrophic rhinosinusitis (A) through a nasal septum perforation shows surrounding crusting and bleeding. (B) The right nasal passage reveals a partial middle turbinectomy with dry mucus crusting and surrounding edema. (C) The left nasal passage reveals a large maxillary antrostomy with dry mucus crusting.
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Rhinoscopic view in atrophic rhinosinusitis through a nasal septum perforation reveals bilateral middle turbinectomies, copious yellow mucopurulent secretion, edema, and minimal crusting.

Rhinoscopic view in atrophic rhinosinusitis through a nasal septum perforation reveals bilateral middle turbinectomies, copious yellow mucopurulent secretion, edema, and minimal crusting.
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Nonkeratinizing squamous metaplasia (arrow) of the normal respiratory epithelium with complete loss of goblet cells and thickening of the basement membrane (arrowhead). The lamina propria contains den

Nonkeratinizing squamous metaplasia (arrow) of the normal respiratory epithelium with complete loss of goblet cells and thickening of the basement membrane (arrowhead). The lamina propria contains dense chronic inflammatory infiltrate composed of lymphocytes and plasma cells. Significant numbers of eosinophils and neutrophils are not identified. Many of the blood vessels in the lamina propria are dilated (asterisk). Original magnification×120.
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Normal respiratory epithelium with areas of the pseudostratified epithelium devoid of goblet cells (arrow) and thickened basement membrane (arrowhead). Original magnification×120.

Normal respiratory epithelium with areas of the pseudostratified epithelium devoid of goblet cells (arrow) and thickened basement membrane (arrowhead). Original magnification×120.
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